Antifungal Pharmaceutical Raw Materials Miconazole Nitrate Raw Powder CAS 22832-87-7
Assay: 99% min
Packing: 25kg/cardboard drum
Character: White crystalline powder.
Usage: Broad-spectrum antifungal pharmaceuticals for the treatment of fungal infections.
Miconazole nitrate is an antifungal pharmaceutical for the treatment of tinea, jock itch, ringworm, athlete's foot, tinea versicolor and fungal paronychia, and by the yeast (eg Candida) and Gram-positive bacterial vaginal infections and secondary infection, otitis externa, bacterial skin infections are also effective.
Biological Activity: Antifungal agent. Inhibits lanosterol demethylase and induces formation of reactive oxygen species.
|Items of analysis
||White or white crystalline powder
||Infrared absorption and standard
||Solution is clear, color is not deep in the standard Y7
||Difficult to dissolve in water, soluble in ethanol, slightly soluble in ethanol
|Loss On Drying
|Assay(On Dry Basis
Anti-inflammatory or antiinflammatory refers to the property of a substance or treatment that reduces inflammation or swelling. Anti-inflammatory drugs make up about half of analgesics, remedying pain by reducing inflammation as opposed to opioids, which affect the central nervous system.
Non-steroidal anti-inflammatory drugs (NSAIDs) alleviate pain by counteracting the cyclooxygenase (COX) enzyme. On its own, COX enzyme synthesizes prostaglandins, creating inflammation. In whole, the NSAIDs prevent the prostaglandins from ever being synthesized, reducing or eliminating the pain.
Some common examples of NSAIDs are aspirin, ibuprofen, and naproxen. The newer specific COX-inhibitors are not classified together with the traditional NSAIDs even though they presumably share the same mode of action.
On the other hand, there are analgesics that are commonly associated with anti-inflammatory drugs but that have no anti-inflammatory effects. An example is paracetamol (known as acetaminophen in the U.S). As opposed to NSAIDs, which reduce pain and inflammation by inhibiting COX enzymes, paracetamol has - as early as 2006 - been shown to block the reuptake of endocannabinoids, which only reduces pain, likely explaining why it has minimal effect on inflammation.
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